Some of my friends -- have dismissed the resperate as quack science. However, before I bought my unit, I researched on the web and came to the conclusion that there is a considerable body of evidence that blood pressure regulation is a very complex mechanism that consists of various nervous and hormonal control systems. One component of the regulatory system of blood pressure is certainly the breathing component. Breathing definitely has short term impact on blood pressure. One might think that it would be related only to short term fluctations in blood pressure, but then the way that long term blood pressure equilibrium is established isn't really understood so entirely possible and as resperate studies seem to indicate there are effects. It be interesting to know what this article came below cam up with -- unfortuntately I don't have access - but maybe someone else out there does.

I been plotting my blood pressure since 2002, and I noticed that my 12 point moving average blood pressure ( normally take 4 measurements a day), shifts up and down often in waves of 3 or 4 days in duration. Peak to valley amplitude will be 10 mmHg. That is it might migrate high for a few days and then dip lower for a few days and then dip higher again. One cannot but help thinking perhaps there some controlling mechanism that creating an almost oscillator pattern to these kinds of things. My personal observation is therefore that there are more components to blood pressure variability than these mayer wave models suggest. There are time of days factors and other cycles that may be more complex than these models think.


Modeling of Mayer waves generation mechanisms
Seydnejad, S.R.; Kitney, R.I.
Engineering in Medicine and Biology Magazine, IEEE
Volume 20, Issue 2, Mar/Apr 2001 Page(s):92 - 100
Digital Object Identifier 10.1109/51.917729
Summary:The observed fluctuations in heart rate and blood pressure are
meaningful rhythmical fluctuations that reflect useful information about
autonomic regulation. These rhythmical fluctuations, known as heart rate
variability (HRV) and blood pressure variation (BPV), are normally grouped
into three major components: (i) the HF component, around 0.25 Hz, in
synchrony with respiratory rate; (ii) the LF component, generally centered
around 0.1 Hz, which is attributed to the sympathetic activity and the
closed-loop controlling action of cardiovascular regulation; (iii) the VLF
component, around 0.04 Hz, which is probably due to the vasorhythmicity
thermoregulatory system or to humoral regulations. Unlike the HF
component, there is still considerable controversy with regard to the
origin of LF and VLF components, the so-called Mayer waves. Finding a
model to represent the LF and VLF components in an appropriate manner,
compatible with the relevant physiology, is the objective of this article.
For this purpose, we first briefly review the underlying physiological
mechanisms. Then, an appropriate mathematical representation for each
mechanism is demonstrated and its performance within a comprehensive model
of the cardiovascular regulatory system is considered. Finally, by
comparing with the experimental results, we evaluate the closeness of the
proposed representations to the actual observations

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